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To post positon annoucements on this board


Please email the following information to:  lipidcorner@asbmb.org

1.  Position title
2.  Position's description
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5.  A date to remove the post or number of weeks you would like the post to stay on-line.

To post positon annoucements on this board


Please email the following information to:  lipidcorner@asbmb.org

1.  Position title
2.  Position's description
3.  Contact information
4.  A logo of your institution (if available)
5.  A date to remove the post or number of weeks you would like the post to stay on-line.

 

 


 

 

 


 

  

 

University of North Carolina – Chapel Hill - Postdoctoral Position

 
A POSTDOCTORAL POSITION at the University of North Carolina – Chapel Hill is available immediately to study the structure, function and metabolism of enzymes of glycerolipid metabolism related to obesity, hepatic steatosis, and/or insulin resistance. Applicants should have a recent PhD in physiology, biochemistry or a related field, a strong interest in mammalian lipid metabolism, an excellent publication record, and experience in one or more of the following topics: mouse energy metabolism and physiology, lipid biochemistry and metabolism, cell biology, or protein structure and function. Our current research asks why an excessive liver content of triacylglycerol impairs hepatic insulin signaling, why the functions and regulation of the isoenzymes of triacylglycerol biosynthesis differ under different physiological conditions, and how proteins interact to channel acyl-CoAs and other lipid intermediates into different metabolic pathways.

Please email your reply with your curriculum vitae, a letter detailing your interests and career plans, and the names of three references to Dr. Rosalind Coleman, University of North Carolina, CB#7461, Chapel Hill, 27599. email: rcoleman@unc.edu. UNC is an Equal Opportunity/Equal Access/Affirmative Action Employer.

Recent publications:

Ellis, JM, Mentock, SB, DePetrillo, MA, Koves, TR, Sen, S, Watkins, SM, Muoio, DM, Cline, GW, Taegtmeyer, H, Shulman, GI, Willis, MS, and Coleman, RA. Mouse cardiac acyl-CoA synthetase-1 deficiency impairs fatty acid oxidation and induces cardiac hypertrophy (2011) Mol. Cell. Biol. 31(6):1252-62. PMID: 21245374

Zhang, C, Wendel, AA, Keogh, MR, Harris, TE, Chen, J, and Coleman, RA. Glycerolipid signals alter mTORC2 to diminish insulin signaling (2012), Proc. Natl. Acad. Sci. USA. Jan 31; 109(5):1667-72. Epub 2012 Jan 17. PMID: 22307628; PMCID: PMC3277174

Klett, EL, Chen, S, Edin, ML, Li, LO, Ilkayeva, O, Zeldin, DC, Newgard, CB, and Coleman, RA. Diminished acyl-CoA synthetase isoform 4 (Acsl4) activity in INS 832/13 cells reduces cellular epoxyeicosatrienoic acid levels and results in impaired glucose-stimulated insulin secretion (2013) J. Biol. Chem. 2013 Jul 26;288(30):21618-29. PMID:23766516; PMCID: PMC3724621

Wendel, AA, Cooper, DE, Ilkayeva, OR, Muoio, DM, and Coleman, RA. Glycerol-3-phosphate acyltransferase (GPAT)-1, but not GPAT4, incorporates newly synthesized fatty acids into triacylglycerol and diminishes fatty acid oxidation. J. Biol. Chem. 288(38):27299-306, 2013. PMID:23908354; PMCID: PMC3779725

Zhang, C, Klett, EL, and Coleman, RA. Lipid signals and insulin resistance. 2013 Clin. Lipidol. 2013, 8: 659-667; PMID:24533033; PMCID:PMC3921899

Paul, DS, Grevengoed, TJ, Pascual, F, Willis, MS, and Coleman, RA. Deficiency of cardiac acyl-CoA synthetase-1 induces diastolic dysfunction, but pathologic hypertrophy is reversed by rapamycin (2014) Biochim. Biophys. Acta. 2014 Mar 12;1841(6):880-887

Li, LO, Grevengoed, TJ, Paul, DS, Pascual, F, Ilkayeva, O, Newgard, CB, Muoio, DM, Coleman, RA. Compartmentalized acyl-CoA metabolism in skeletal muscle compromises systemic glucose homeostasis (2014) Diabetes, Jul 28. pii: DB_131070. [Epub ahead of print]; PMID:25071025

 

Posted: 8-11-2014

 

University of North Carolina – Chapel Hill - Postdoctoral Position

 
A POSTDOCTORAL POSITION at the University of North Carolina – Chapel Hill is available immediately to study the structure, function and metabolism of enzymes of glycerolipid metabolism related to obesity, hepatic steatosis, and/or insulin resistance. Applicants should have a recent PhD in physiology, biochemistry or a related field, a strong interest in mammalian lipid metabolism, an excellent publication record, and experience in one or more of the following topics: mouse energy metabolism and physiology, lipid biochemistry and metabolism, cell biology, or protein structure and function. Our current research asks why an excessive liver content of triacylglycerol impairs hepatic insulin signaling, why the functions and regulation of the isoenzymes of triacylglycerol biosynthesis differ under different physiological conditions, and how proteins interact to channel acyl-CoAs and other lipid intermediates into different metabolic pathways.

Please email your reply with your curriculum vitae, a letter detailing your interests and career plans, and the names of three references to Dr. Rosalind Coleman, University of North Carolina, CB#7461, Chapel Hill, 27599. email: rcoleman@unc.edu. UNC is an Equal Opportunity/Equal Access/Affirmative Action Employer.

Recent publications:

Ellis, JM, Mentock, SB, DePetrillo, MA, Koves, TR, Sen, S, Watkins, SM, Muoio, DM, Cline, GW, Taegtmeyer, H, Shulman, GI, Willis, MS, and Coleman, RA. Mouse cardiac acyl-CoA synthetase-1 deficiency impairs fatty acid oxidation and induces cardiac hypertrophy (2011) Mol. Cell. Biol. 31(6):1252-62. PMID: 21245374

Zhang, C, Wendel, AA, Keogh, MR, Harris, TE, Chen, J, and Coleman, RA. Glycerolipid signals alter mTORC2 to diminish insulin signaling (2012), Proc. Natl. Acad. Sci. USA. Jan 31; 109(5):1667-72. Epub 2012 Jan 17. PMID: 22307628; PMCID: PMC3277174

Klett, EL, Chen, S, Edin, ML, Li, LO, Ilkayeva, O, Zeldin, DC, Newgard, CB, and Coleman, RA. Diminished acyl-CoA synthetase isoform 4 (Acsl4) activity in INS 832/13 cells reduces cellular epoxyeicosatrienoic acid levels and results in impaired glucose-stimulated insulin secretion (2013) J. Biol. Chem. 2013 Jul 26;288(30):21618-29. PMID:23766516; PMCID: PMC3724621

Wendel, AA, Cooper, DE, Ilkayeva, OR, Muoio, DM, and Coleman, RA. Glycerol-3-phosphate acyltransferase (GPAT)-1, but not GPAT4, incorporates newly synthesized fatty acids into triacylglycerol and diminishes fatty acid oxidation. J. Biol. Chem. 288(38):27299-306, 2013. PMID:23908354; PMCID: PMC3779725

Zhang, C, Klett, EL, and Coleman, RA. Lipid signals and insulin resistance. 2013 Clin. Lipidol. 2013, 8: 659-667; PMID:24533033; PMCID:PMC3921899

Paul, DS, Grevengoed, TJ, Pascual, F, Willis, MS, and Coleman, RA. Deficiency of cardiac acyl-CoA synthetase-1 induces diastolic dysfunction, but pathologic hypertrophy is reversed by rapamycin (2014) Biochim. Biophys. Acta. 2014 Mar 12;1841(6):880-887

Li, LO, Grevengoed, TJ, Paul, DS, Pascual, F, Ilkayeva, O, Newgard, CB, Muoio, DM, Coleman, RA. Compartmentalized acyl-CoA metabolism in skeletal muscle compromises systemic glucose homeostasis (2014) Diabetes, Jul 28. pii: DB_131070. [Epub ahead of print]; PMID:25071025

 

Posted: 8-11-2014